
Publikationen von Heinrich Schrewe
Alle Typen
Zeitschriftenartikel (9)
2010
Zeitschriftenartikel
177 (2), S. 840 - 853 (2010)
Long-Term Expression of Tissue-Inhibitor of Matrix Metalloproteinase-1 in the Murine Central Nervous System Does Not Alter the Morphological and Behavioral Phenotype but Alleviates the Course of Experimental Allergic Encephalomyelitis. The American Journal of Pathology
Zeitschriftenartikel
177 (2), S. 840 - 853 (2010)
Long-Term Expression of Tissue-Inhibitor of Matrix Metalloproteinase-1 in the Murine Central Nervous System Does Not Alter the Morphological and Behavioral Phenotype but Alleviates the Course of Experimental Allergic Encephalomyelitis. The American Journal of Pathology 2003
Zeitschriftenartikel
197 (1), S. 149 - 156 (2003)
Sorsby Fundus Dystrophy Mutation Timp3S156C Affects the Morphological and Biochemical Phenotype But Not Metalloproteinase Homeostasis. Journal of Cellular Physiology
Zeitschriftenartikel
23 (1), S. 272 - 279 (2003)
Carcinoembryonic Antigen-Related Cell Adhesion Molecule 10 Expressed Specifically Early in Pregnancy in the Decidua is Dispensable for Normal Murine Development. Molecular and Cellular Biology 2002
Zeitschriftenartikel
16 (21), S. 2749 - 2754 (2002)
Activin type IIA and IIB receptors mediate Gdf11 signaling in axial vertebral patterning. Genes & Development
Zeitschriftenartikel
43 (8), S. 2732 - 2740 (2002)
A Mouse Model for Sorsby Fundus Dystrophy. Investigative Ophthalmology & Visual Science
Zeitschriftenartikel
99 (9), S. 6222 - 6227 (2002)
Inactivation of the murine X-linked juvenile retinoschisis gene, Rs1h, suggests a role of retinoschisin in retinal cell layer organization and synaptic structure. Proceedings of the National Academy of Sciences of the United States of America
Zeitschriftenartikel
112 (1-2), S. 69 - 77 (2002)
Nuclear factor I-B (Nfib) deficient mice have severe lung hypoplasia. Mechanisms of Development
Zeitschriftenartikel
32 (2 Sp. Iss. SI), S. 121 - 123 (2002)
Conditional Inactivation of Sox9: A Mouse Model for Campomelic Dysplasia. Genesis